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If cancer is a disease precipitated by changes in genes, after all, we need to know lots about how and when different genes change in the many distinctive subtypes of cancer.
But a new wave of research, exemplified by a study published in Nature Genetics by a team at Cold Spring Harbor Laboratory (CSHL), is significantly improving our ability to target cancer cells by studying "the other 98%" of DNA in human chromosomes, sometimes called the genome's "dark matter." Research led by Michael Feigin, Ph.
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While the promoter mutations were not near known pancreatic cancer genes, the team found that they affected some of the same biological pathways in cells.
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